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Preface

Summary

1. Timeline

2. Virology

3. Transmission

4. Epidemiology

5. Prevention

6. Case Definition

7. Diagnostic Tests

8. Clinical Presentation and Diagnosis

9. SARS Treatment

10. Pediatric SARS



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9. SARS Treatment Loletta Kit-Ying SO ( Correspondence)
Arthur Chun-Wing LAU
Loretta Yin-Chun YAM The treatment of coronavirus-associated SARS has been evolving and so far there is no consensus on an optimal regimen. This chapter reviews the diverse treatment experience and controversies to date, and aims to consolidate our current knowledge and prepare for a possible resurgence of the disease.

Results  One patient from the corticosteroid group was excluded from analyses because of consent withdrawal. There were 229 nonresponders to the corticotropin test (placebo, 115; corticosteroids, 114) and 70 responders to the corticotropin test (placebo, 34; corticosteroids, 36). In nonresponders, there were 73 deaths (63%) in the placebo group and 60 deaths (53%) in the corticosteroid group (hazard ratio, ; 95% confidence interval, -; P = .02). Vasopressor therapy was withdrawn within 28 days in 46 patients (40%) in the placebo group and in 65 patients (57%) in the corticosteroid group (hazard ratio, ; 95% confidence interval, -; P = .001). There was no significant difference between groups in responders. Adverse events rates were similar in the 2 groups.

Dermatitis affected about 10% of . workers in 2010, representing over 15 million workers with dermatitis. Prevalence rates were higher among females than among males, and among those with some college education or a college degree compared to those with a high school diploma or less. Workers employed in healthcare and social assistance industries and life, physical, and social science occupations had the highest rates of reported dermatitis. About 6% of dermatitis cases among . workers were attributed to work by a healthcare professional, indicating that the prevalence rate of work-related dermatitis among workers was at least %. [70]

Etomidate is usually presented as a clear colourless solution for injection containing 2 mg/ml of etomidate in an aqueous solution of 35% propylene glycol , although a lipid emulsion preparation (of equivalent strength) has also been introduced. Etomidate was originally formulated as a racemic mixture, [39] but the R form is substantially more active than its enantiomer . [40] It was later reformulated as a single-enantiomer drug, becoming the first general anesthetic in that class to be used clinically. [41]

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Etomidate is usually presented as a clear colourless solution for injection containing 2 mg/ml of etomidate in an aqueous solution of 35% propylene glycol , although a lipid emulsion preparation (of equivalent strength) has also been introduced. Etomidate was originally formulated as a racemic mixture, [39] but the R form is substantially more active than its enantiomer . [40] It was later reformulated as a single-enantiomer drug, becoming the first general anesthetic in that class to be used clinically. [41]

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